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Julie Atkin

Professor

Former affiliations

Macquarie University

 https://orcid.org/0000-0003-2427-499X
  • 0 Citations
  • 0 h-Index
20052020

Research output per year

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Personal profile

Biography

Julie is interested in the molecular and cellular mechanisms that trigger neurodegeneration in amyotrophic lateral sclerosis (ALS).

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Research Output

The redox activity of protein disulfide isomerase inhibits ALS phenotypes in cellular and zebrafish models

Parakh, S., Shadfar, S., Perri, E. R., Ragagnin, A. M. G., Piattoni, C. V., Fogolín, M. B., Yuan, K. C., Shahheydari, H., Don, E. K., Thomas, C. J., Hong, Y., Comini, M. A., Laird, A. S., Spencer, D. M. & Atkin, J. D., 22 May 2020, In : iScience. 23, 5, p. 1-27 44 p., 101097.

Macquarie University

Research output: Contribution to journal › Article › Research › peer-review

Open Access

    • Amyotrophic lateral sclerosis-linked UBQLN2 mutants inhibit endoplasmic reticulum to Golgi transport, leading to Golgi fragmentation and ER stress

    Halloran, M., Ragagnin, A. M. G., Vidal, M., Parakh, S., Yang, S., Heng, B., Grima, N., Shahheydari, H., Soo, K-Y., Blair, I., Guillemin, G. J., Sundaramoorthy, V. & Atkin, J. D., 4 Dec 2019, In : Cellular and Molecular Life Sciences. 77, p. 3859-3873 15 p.

    Macquarie University

    Research output: Contribution to journal › Article › Research › peer-review

    • Pathogenic mutation in the ALS/FTD gene, CCNF, causes elevated Lys48-linked ubiquitylation and defective autophagy.

    Lee, A., Rayner, S. L., Gwee, S. S. L., De Luca, A., Shahheydari, H., Sundaramoorthy, V., Morsch, M., Hogan, A., Don, E., Williams, K., Yerbury, J. J., Blair, I., Atkin, J. D., Molloy, M. P. & Chung, R. S., 8 Feb 2019. 1 p.

    Macquarie University

    Research output: Contribution to conference › Abstract › Research › peer-review

    • Structural requirements for the interaction of human IgA with the human polymeric Ig receptor

    Lewis, M. J., Pleass, R. J., Batten, M. R., Atkin, J. D. & Woof, J. M., 15 Nov 2005, In : Journal of Immunology. 175, 10, p. 6694-6701 8 p.

    Research output: Contribution to journal › Article › Research › peer-review